Pancreatitis and irritable bowel syndrome


IBS or chronic pancreatitis?

Ivan IvanovPupil (6), closed 1 year ago


After alcohol, I start to puchit, I go to the toilet 2 times
a day, sometimes a gruel, but not diarrhea. There are no pains, but I feel discomfort
in the intestine. Day after 2-3 it passes. For other food reactions no
it happens - neither greasy nor sharp.

Went to the doctor, she felt everything, nothing
found, sent to hand over the feces. The analysis is ideal, the diagnosis is skk. I wanted to make uzi
pancreas or fgs, but she did not say, and that chronic pancreatitis is not
can flow without pain.

Have I been diagnosed correctly or are
continue to walk on the doctors?

And do not say that you should drink less. I'm already drinking
maximum once every 2 weeks, a gram of 200, and then not always.

♍Galina Zhigunova♍Supreme Mind (830354) 3 years ago

I have chronic pancreatitis, there never is pain. At its or his aggravation terrible nausea and vomiting open.


Here is his clinic:
Symptoms, course. Pain in the epigastric region and the left hypochondrium, dyspepsia, diarrhea, weight loss, diabetes mellitus. The pain is localized in the epigastric region to the right with the primary localization of the process in the region of the head of the pancreas, when involved in the inflammatory process of its body - in epigastric region on the left, with the defeat of its tail in the left hypochondrium; often the pain irradiates in the back and has a shrouding character, can irradiate into the heart area, imitating angina pectoris. Pain can be permanent or paroxysmal and appear after a while after taking fatty or spicy food. There is a pain in the epigastric region and the left hypochondrium. Often there is a painful point in the left costal-vertebral corner (the Mayo-Robson symptom). Sometimes the zone of cutaneous hyperesthesia is determined, respectively, in the innervation zone of the eighth thoracic segment on the left (symptom Kacha) and some atrophy of subcutaneous adipose tissue in the area of ​​the projection of the pancreas on the front wall stomach.

Dyspeptic symptoms in chronic pancreatitis are almost constant. Frequent loss of appetite and aversion to fatty foods are common. However, when developing diabetes, on the contrary, patients can feel strong hunger and thirst. Often observed increased salivation, belching, attacks of nausea, vomiting, flatulence, rumbling in the abdomen. Stools in mild cases are normal, in more severe cases - diarrhea or alternating constipation and diarrhea. Characteristic of pancreatic diarrhea with the release of a plentiful, mildew-like stench with a greasy shine; A scatological research reveals steatori, creator, and kitarinoreyu.

777ооо777Guru (3192) 3 years ago

Well, as you like. Any dose of alcohol has an effect on the liver and gallstones. Peat Atoxil, activated carbon, and more simple boiling water.

IBS or Chronic pancreatitis? - Forum for Patients with Pancreatitis

Status: Not available

Hello. I am 25 years old. I want to tell my story of ailment. In the year I got addicted to fast food - McDonald's and stuff. Not abused. Basically, he leaned after work in the night shift in the bar 1-2 times a week for 2-3 months. The work was tense, jaundiced. I eat mostly correctly. Fat and fried I rarely use. I do not drink or smoke. In one of these night feasts I felt a sharp sharp pain in the left hypochondrium. It took 15-20 minutes. Did not pay attention to it. Since then, after consuming a lot of food, for example, at dinner, and having drunk a glass of cha, overflow in the abdomen and a slight acute pain in the left hypochondrium, accompanied by belching and flatulence. Most of the pain appeared with a deep breath. If you did not eat a lot, there was not any. In m he replaced the place of work. I fastened with fast food. The regime as a whole recovered. But the pain was also present after eating. Plus began to notice flatulence and eructations after eating. All this time, and in general as a whole during the years 10-15 the chair was mostly with constipation. Diarrhea did not arise. In March, he decided to be examined. I started with fgd, .k. I think, my stomach hurts after eating. Fgds in the norm (photo attached on the link). Then he was at a reception with a gastroenterologist. Told the complaint. Was directed to ultrasound of the abdominal cavity and irrigoscopy. Preliminary diagnosis - IBS. Irrigoscopy did not. The ultrasound has done. Conclusion on ultrasound: Diffuse pancreatic changes. Constrictions of the gallbladder. Another doctor, the Uzzyst skaz, then in the bilious is either poly, for the stone. Has subscribed to other gastroenterologist. He showed the results of uzi and phlogs. He was diagnosed with chronic pancreatitis. Have appointed Pancreatin, Ganaton, Dufalac and diet 5n. The month was treated. Symptoms did not go away, except for acute pain, .k. began to eat less and keep to a diet. All the same flatulence, heaviness in the left hypochondrium with a deep breath. Constipation continued. I went to the toilet once every three days. But notice, after the act of defecation on toilet paper, traces of a drop of red blood began to remain. I registered with the proctologist. The proctologist did not find anything serious. Say, I consume a little liquid and vegetables. And the blood is due to tog, the mucous is injured by dry calves due to constipation and fluid deficiency. Received recommendations and treatment of constipation (abundant to drink, more cellulose, orlax). He started treatment. After taking forlax within 3 days, there was no improvement. On the 4th day, diarrhea began. The chair was unformed, mushy, laval on the surface of the water, chalk yellow-brown, stew greasy film on the surface of the water, poorly washed off and there were visible particles of undigested cellulose. Immediately I stopped taking forlax. But since then, after every meal for an hour, meteoritis, a slight dull pain in the left podreber, a jug and a plentiful mushy stool, described above. I decided to renew the method of pancreatin and notice, then the chair began to normalize. Continued to follow the diet of the proctologist within a week. Forlax stopped drinking. It became worse. Pancreatin did not help. For 2 weeks I lost 10 kg. Again came to the third gastroenterologist, because I wanted to learn as much as possible opinions about this condition. The gastroenterologist has diagnosed - a chronic pancreatitis in a stage of remission. Assigned kreon 10000, chamomile broth, olpazu and one-time tjubazh. Was treated for 2 more weeks. The condition became better. Pains after eating became less, about feces and progressive weight loss with flatulence and belching remained. Increased the dose of Creon to 2, 00. The condition became better. I went to see a therapist with a request to be examined in a gastroenterological hospital. Before they went to the hospital, he submitted a feces for a coprogram and a general blood test. (photo attached on the link). Cal showed the presence of digested and undigested muscle fibers, undigested plant cells, the appearance of neutral fat, the difference between starch. For the sake of interest he opened a children's card and found 3 coprograms for the last 17 years. On all coprograms were the result, then I described the above, rum availability of starch. There was no starch. The blood showed ALT-3, CT-2, or ruby ​​common-straight-2, Amelase-7, lucose, total protein-74. In the hospital, he received a course of treatment with metronidazole, drotaverin, glucose, metoclopramido, meprazole and ciprofloxacin. During a 2-week survey, on the recommendation of a doctor, I took pancroz 2, 00 ed 3 times a day. The following studies were performed: FGD, program, ZI OB, ectomanoscopy, urinalysis, iochemical analysis of blood and ECG. After 2 weeks, the condition returned to normal. The pains disappeared. Blown up, theories have disappeared. The eructation was very rare. The coprogram became good. A diagnosis was made of chronic colic, tadia exacerbation, ronified gastritis, tadia exacerbation and IBS. It was prescribed treatment with trimedato, iofloro balance and pangrolom 25000 units 3p per day during the week. A week later, he stopped taking Pangrol 2, 00 on recommendation. And he continued treatment with trimedate and rioflora. Two days after the cessation of taking Pangrol all symptom, they were returned during the last two months. After eating for an hour in the toilet. BUT! Desires for defecation do not occur after each meal, as soon as possible. Depends on the amount eaten. If you eat half the proportion - nothing. Or a small meteorosis, a jerk and a false urge to defecate. If complete - a meteorosis, a jerk and desires on a defecation with the subsequent emptying of an intestine. Cal are profuse, irish and unformed. Already dull not severe pain in the left hypochondrium with a deep breath and to the left of the navel. And the continuing weight loss. Tell or say pozhalujst, whether ozhet such status to be caused or called because of sharp stopping of reception of enzymes? And is the conclusion of the gastroenterological hospital correct? I want to conduct an independent diagnosis and submit an analysis, recommended in this forum.

But from this list in our locality I will be able to pass tests only on:
fecal elastase-1.
isoenzymes of amylase, C-reactive protein and CT of the prostate.

Tell or say pozhalujst, silent analyzes will be enough for statement of the exact diagnosis?
Maybe l, then I really have CP, not HP? And how do you think this preliminary diagnosis can be made from this anamnesis? My opinion is the inauthenticity and inadequacy of the analyzes on HP. Because the conclusion was made on the absence of symptoms and coprogram. But the coprogram depends on the enzymes. On uzi nothing was found, except for a constriction of a cholic bubble. Uzzisty with a difference of 3 months were different.

Message edited by Dreamdau5 - Воскресенье, 31.07., 2: 4

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Irritable Bowel Syndrome

Irritable Bowel Syndrome (IBS) is a polyethiologic disease that has a chronic course and significantly impairs the quality of life of patients. Approximately one third of the population suffers from these violations.

It is worth noting that there is no absolutely accurate description of the characteristics of IBS. Currently used are the so-called # 17; the Romanized criteria of modernization # 187; 2000 year. They sound like: discomfort in the abdomen, including stomach pain. duration of at least 12 weeks, but not more than 12 months. In this case there should be two signs out of three, namely: reduction of pain syndrome, change in stool frequency, physical properties of feces.

IBS is characterized by multiple functional pathologies of the motor function, disturbances in the processes digestion, absorption of food components in the large intestine, as well as changes in the secretion of cells intestines. Multiple histological studies in irritable bowel syndrome suggest that there are no obvious organic disorders of the intestinal wall state.

The exact causes of the disease are unknown. Rather, it is multiple factors, both internal and external. From exogenous factors it is necessary to allocate:

  • poor-quality and irregular meals;
  • food with a predominance of high-calorie food;
  • the use of various food additives;
  • alcohol consumption;
  • excess fat and cholesterol, as well as concentrated protein components in foods;
  • excessive consumption of caffeine and corrosive substances contained in carbonated beverages.

A large influence is exerted by unfavorable environmental factors (temperature and pressure drops, external radiation, electromagnetic media).

It is noticed that more often the disease occurs in people with chronic neurological disorders, suffering from insomnia and depression.

From the endogenous causes of development it should be noted: various unexplained dysfunctions of central and vegetative nervous systems, stresses of various genesis, dysbacteriosis and other disorders in the microbiological composition of tissues organism. Symptoms of IBS are mostly registered in patients who have some kind of abnormality from the endocrine system.

In our opinion, the most significant reason for the appearance of the pathological process is the presence of chronic pancreatitis with a violation of exocrine function, which leads to the launch of mechanisms of pathological digestion food. Also, the development of IBS is greatly influenced by functional disorders in the activity of the gallbladder.

It is impossible to exclude the possibility that the triggering mechanism of irritable bowel syndrome can be fermentopathy # 8211; congenital absence of this or that protein complex, which is secreted by cells of the gastrointestinal tract for digestion of food.



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